High cholesterol has received a bad rap for prolonged poor heart health-but a new study suggests that low carb ketogenic diet cannot be connected to heart disease.
The study led by Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center in collaboration with researchers in many institutes assessed 100 participants after a long-term Keto diet, who developed high levels of LDL cholesterol (known as “poor” types).
In addition to high cholesterol, all participants were “healthy with metabolism” and according to the April 7 press release, followed the major diet for five years on an average.
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They are all eligible as LMHR (lean mass hyper-raper-ripper), indicating those who adopt carbohydrate-stuffed diet and experience a significant increase in cholesterol.
High cholesterol has received a bad rap for prolonged poor heart health-but a new study suggests that low carb ketogenic diet cannot be connected to heart disease. (Istock)
Using advanced cardiac imaging, researchers found that traditional cholesterol markers (APOB and LDL-C) were not associated with plaque levels in the arteries of the heart or baseline heart disease over a period of one year.
Instead, the existing plaque level appears to be a better predictive of future plaque accumulation.
“This population of people – being in ketosis is healthy with metabolism with high LDL – automatically does not increase cardiac risk, as their LDL is elevated,” Baszuki Group’s medical director Brett Sher, who provided funding for studies, told Fox News Digital.
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The California-based doctor said, “Therefore, we are likely to move away from LDL and APOB and for a better risk predicting how to treat vascular imaging with CAC or CTA and how to treat someone’s heart risk factors or if someone’s cardiac risk factors should be treated,” California-based doctor said.
The findings of the study were published in American College of Cardiology: Advanced Journal.
Using advanced cardiac imaging, researchers found that traditional cholesterol markers (APOB and LDL-C) were not associated with plaque levels in the arteries of the heart or baseline heart disease over a period of one year. (Istock)
Previous studies have also shown that people who qualify as LMHR have the same levels of coronary plaque, otherwise comparable groups have normal LDL levels, “saying that the ketogenic diet-inspired LDL growth cannot indicate a high risk of the coronary plaque, saying that the researcher said.
The leader and independent researcher of a study at Oxford University, Dr. Nick Norwitz stated that this is the first study to separate LDL and APOB as risk factors of heart disease.
“All other human studies include individuals with metabolic dysfunction or innate genetic reasons of high LDL,” he told Fox News Digital.
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According to Norwitz, what most doctors would have predicted and doctors are taught in medical training, the result appears.
“While these data do not prove that the traditional understanding is’ wrong,” they suggest that there is a big blind place in the traditional model. “
This is the first potential test of its kind in a unique population that is often labeled ‘high-risk’ by traditional guidelines, a doctor said, raising important questions about how the heart risk is evaluated in the context of low carb, high-fat diet. (Istock)
According to Norwitz, cardiac imaging, including CAC scores, is “more value” than cholesterol levels in predicting plaque progression.
“Thus, the CAC score can be used to help patients to risk and make care individual,” he told Fox News Digital.
Scher said that “ketogenic therapy” may be effective in treating some metabolic conditions, but some people are afraid to continue diets due to their cholesterol.
“This study provides support that they do not need to prevent diet or treat their cholesterol – rather, they can work with their healthcare team for more personal and proper cardiac workups,” they advised.
Raise important questions
Dr. Ken Berry, a family doctor and Diabetic specialist In Tennessee, research was not involved, but shared his views that whatever he described as “groundbreaking” studies.
“The study found no connection between the progression of LDL-C, APOB and coronary plaque, which is a year in a year using high-resolution CT angiography,” he asked Fox News Digital.
“Instead, the strongest prophet of plaque progression was the already existing plaque, not at the cholesterol level-the sufficient researchers concluded that ‘the plaque forgets the plaque, not APOB.”
The existing plaque level seemed to be a better prophet of future plaque accumulation, although some experts warned that boundaries exist. (Istock)
This is often the first possible test of its kind in a unique population of ‘high-risk’ labeled by traditional guidelines, Berry said, raising important questions about how the heart risk is evaluated in the context of low carb, high-fat diet.
“The clear implication is that if this specific group of too much APOB level is not a good prophet of the risk of heart attack, is it a good prophet in any group of people?” He said.
“Or is it, as I doubt, just the latest popular laboratory tests are being used to remove people from eating a proper human diet rich in saturated fat?”
Potential study limitations
Dr. Bradley Serwar, a cardiologist and Chief Medical Officer who is a Cincinnati-based company, which provides heart and anesthesiology services in nationwide hospitals, reviewed the study and indicated some possible limitations.
“Limited scope of study, incorporating low -risk population in a short period, is challenging to normalize conclusions for a broad, more weaker population,” Serwar explained Fox News Digital.
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“While the purpose of the study was to propose a hypothesis about the role of diet cholesterol, it does not provide certain evidence for or for its importance.”
Cardiologist, however, agrees with the finders’ findings that “better risk stratification tool” are necessary to identify high risk individuals of coronary artery disease.
The effect of ketogenic diet can be measured using ketosis meters. “I hope doctors will embrace this research and treat this specific population of people differently from the rest of their patients, providing the unique physical condition and metabolic benefits of ketosis,” said a researcher said. (Istock)
“As physicians, our primary responsibility lies to develop every patient to evaluate on individual basis and develop the most suitable long -term care plan in collaborating with them through shared decision making,” he said.
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Michelle Ruthinstein registered dietist, who specializes in heart disease in New York City, said that plaque formation is a multistap process that may take years to progress.
“The environment of the artery must be conducive to plaque formation,” Ruthnestine, who was not part of the study, told Fox News Digital.
“It is a remarkable group of humans showing remarkable physiology.”
“For example, individuals with high blood pressure, a subgroup that was excluded from the study, is more prone to endothelial damage that can cause APOB to accumulate more easily into the artery wall.”
“If someone already has a plaque in the arteries and maintains a high level of LDL and APOB, it may develop into a higher plaque, as seen in this study.”
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“However, if anyone is healthy with metabolism, there is no plaque on the baseline, and alone APOB and LDL levels have elevated, the environment may not necessarily cause plaque over a period of one year.”
Routhentein showed prior research that high LDL and APOB in the years of one’s life, usually with swelling, insulin resistance and/or oxidative stress, may increase the risk of plaque development.
“It is important to note that many people who are applying to -ogenic diet and ignoring high LDL and APOB levels are usually not knowing that they have a soft plaque.” (Istock)
He said, “It is important to note that many people who are implementing the ketogenic diet and ignoring high LDL and APOB levels are usually not knowing that they have a soft plaque.”
“Therefore, it can be harmful to advise them to ignore LDL and APOB levels – especially in a world where heart disease is so prevalent and is the leading cause of death globally.”
looking ahead
Lion said he hopes that more researchers would be motivated to pursue this study and apply it to different population.
“But for now, I hope that doctors will embrace this research and treat this specific population of people differently from the rest of their patients, providing the unique physiological status and metabolic benefits of ketosis,” he said.
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In addition to more studies assessing the risk in this population, Norwitz said that the team hopes to examine the mechanism of lean mass hyper-ripper (LMHR) phenotypes.
“It is a notable group of humans that demonstrate remarkable physiology,” he said.
